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Year : 2019  |  Volume : 16  |  Issue : 2  |  Page : 93-96

Skull-base fractures: Pearls of etiopathology, approaches, management, and outcome

1 Department of Neurosurgery, National Institute of Mental Health and Neuro-Sciences, Bengaluru, Karnataka, India
2 Centro de Investigaciones Biomédicas, University of Cartagena, Cartagena de Indias, Bogota, Colombia
3 RED LATINO, Organización Latinoamericana de Trauma y Cuidado Neurointensivo, Bogota, Colombia
4 Department of Neurosurgery, All India Institute of Medical Sciences, New Delhi, India
5 Department of Anesthesia, Narayana Medical College Hospital, Nellore, Andhra Pradesh, India
6 Department of Community Medicine, MGM Medical College and LSK Hospital, Kishanganj, Bihar, India
7 Department of Neurosurgery, Narayana Medical College Hospital, Nellore, Andhra Pradesh, India

Correspondence Address:
Amit Agrawal
Department of Neurosurgery, Narayana Medical College Hospital, Chinthareddy Palem, Nellore - 524 003, Andhra Pradesh
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Source of Support: None, Conflict of Interest: None

DOI: 10.4103/am.am_12_19

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Fractures involving skull bases usually results from high-velocity impacts, i.e., motor vehicular accidents. The estimated incidence of skull-base fracture varies from 4% of all head injuries to 7%–16% of closed head injuries. Many studies have emphasized the importance of the management of basal skull fractures as it can be associated with major neurological deficits, can carry a risk of being missed during clinical evaluation in a busy emergency room and can additionally lead to life-threatening complications including cerebrospinal fluid (CSF) leak and meningitis. In addition to the bony injuries, skull-base fractures can be accompanied by injury to cranial nerves, intracranial blood vessels, and CSF rhinorrhea or otorrhea (particularly fractures involving anterior and middle cranial fossa). In the presence of characteristics clinical features, patient should be thoroughly clinical examination and pertinent investigation with appropriate imaging modality for the presence of additional intracranial lesions and as delayed diagnosis can lead to increase in neurological morbidity and also rarely mortality.

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